Please use this identifier to cite or link to this item: http://hdl.handle.net/11452/23268
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dc.contributor.authorTvarijonaviciute, Asta-
dc.contributor.authorCeron, Jose J.-
dc.date.accessioned2021-12-15T08:39:57Z-
dc.date.available2021-12-15T08:39:57Z-
dc.date.issued2011-03-15-
dc.identifier.citationTvarijonaviciute, A. vd. (2011). "Adiponectin and IGF-1 are negative acute phase proteins in a dog model of acute endotoxaemia". Veterinary Immunology and Immunopathology, 140(1-2), 147-151.en_US
dc.identifier.issn0165-2427-
dc.identifier.issn1873-2534-
dc.identifier.urihttps://doi.org/10.1016/j.vetimm.2010.11.011-
dc.identifier.urihttps://www.sciencedirect.com/science/article/pii/S0165242710003934-
dc.identifier.urihttp://hdl.handle.net/11452/23268-
dc.description.abstractThe objective of this study was to evaluate the influence of an experimentally induced acute inflammation on serum adiponectin and insulin-like growth factor 1 (IGF-1) levels in the dog, and to compare their evolution with other well-established acute phase proteins (APPs) such as C-reactive protein (CRP), and haptoglobin (Hp). Therefore levels of adiponectin, IGF-1 and a profile of APPs were measured in healthy dogs after intravenous administration of E. coli LPS (0.02 mg/kg) and compared with dogs injected with saline solution (0.2 mL/kg). Adiponectin and IGF-1 were both decreased in response to endotoxins in the dog. Significant positive correlations were found between adiponectin and IGF-1 (r = 0.31; p < 0.05). Adiponectin had also a significant negative correlation with CRP (r = 0.39; p < 0.05) and Hp (r = -0.27; p < 0.05), whereas IGF-1 had significant negative correlation with CRP (r = -0.52; p < 0.001). The results obtained in the present study indicate that adiponectin and IGF-1 behave as negative acute phase proteins after acute inflammatory stimulus in dogs.en_US
dc.language.isoenen_US
dc.publisherElsevieren_US
dc.rightsinfo:eu-repo/semantics/closedAccessen_US
dc.subjectImmunologyen_US
dc.subjectVeterinary sciencesen_US
dc.subjectAdiponectinen_US
dc.subjectCRPen_US
dc.subjectHpen_US
dc.subjectDogen_US
dc.subjectEndotoxaemiaen_US
dc.subjectIGF-1en_US
dc.subjectExpressionen_US
dc.subjectInflammationen_US
dc.subjectDecreaseen_US
dc.subjectCholineen_US
dc.subjectInjuryen_US
dc.subjectCanis familiarisen_US
dc.subject.meshAdiponectinen_US
dc.subject.meshAnimalsen_US
dc.subject.meshC-reactive proteinen_US
dc.subject.meshDisease models, animalen_US
dc.subject.meshDog diseasesen_US
dc.subject.meshDogsen_US
dc.subject.meshEndotoxemiaen_US
dc.subject.meshEscherichia colien_US
dc.subject.meshFemaleen_US
dc.subject.meshHaptoglobinsen_US
dc.subject.meshInsulin-like growth factor ien_US
dc.subject.meshLipopolysaccharidesen_US
dc.subject.meshMaleen_US
dc.titleAdiponectin and IGF-1 are negative acute phase proteins in a dog model of acute endotoxaemiaen_US
dc.typeArticleen_US
dc.identifier.wos000288342800018tr_TR
dc.identifier.scopus2-s2.0-79960988651tr_TR
dc.relation.publicationcategoryMakale - Uluslararası Hakemli Dergitr_TR
dc.contributor.departmentUludağ Üniversitesi/Veterinerlik Fakültesi/Dahiliye Anabilim Dalı.tr_TR
dc.contributor.orcid0000-0002-4242-8609tr_TR
dc.contributor.orcid0000-0001-9836-0749tr_TR
dc.identifier.startpage147tr_TR
dc.identifier.endpage151tr_TR
dc.identifier.volume140tr_TR
dc.identifier.issue1-2tr_TR
dc.relation.journalVeterinary Immunology and Immunopathologyen_US
dc.contributor.buuauthorEralp, Oya İnan-
dc.contributor.buuauthorKocatürk, Meriç-
dc.contributor.buuauthorYılmaz, Zeki-
dc.contributor.researcheridAAG-2943-2020tr_TR
dc.relation.collaborationYurt dışıtr_TR
dc.identifier.pubmed21145113tr_TR
dc.subject.wosImmunologyen_US
dc.subject.wosVeterinary sciencesen_US
dc.indexed.wosSCIEen_US
dc.indexed.scopusScopusen_US
dc.indexed.pubmedPubmeden_US
dc.wos.quartileQ3 (Immunology)en_US
dc.wos.quartileQ1 (Veterinary sciences)en_US
dc.contributor.scopusid24472964600tr_TR
dc.contributor.scopusid36437200800tr_TR
dc.contributor.scopusid35944810500tr_TR
dc.subject.scopusAdiponectin Receptors; AdipoRon; Hypoadiponectinemiaen_US
dc.subject.emtreeAcute phase proteinen_US
dc.subject.emtreeC reactive proteinen_US
dc.subject.emtreeEscherichia coli endotoxinen_US
dc.subject.emtreeHaptoglobinen_US
dc.subject.emtreeLipopolysaccharideen_US
dc.subject.emtreeSodium chlorideen_US
dc.subject.emtreeAdiponectinen_US
dc.subject.emtreeC reactive proteinen_US
dc.subject.emtreeHaptoglobinen_US
dc.subject.emtreeLipopolysaccharideen_US
dc.subject.emtreeSomatomedin Cen_US
dc.subject.emtreeAcute endotoxemiaen_US
dc.subject.emtreeAnimal experimenten_US
dc.subject.emtreeArticleen_US
dc.subject.emtreeControlled studyen_US
dc.subject.emtreeDogen_US
dc.subject.emtreeEndotoxemiaen_US
dc.subject.emtreeEscherichia colien_US
dc.subject.emtreeFemaleen_US
dc.subject.emtreeImmunostimulationen_US
dc.subject.emtreeInflammationen_US
dc.subject.emtreeMaleen_US
dc.subject.emtreeNonhumanen_US
dc.subject.emtreeProtein blood levelen_US
dc.subject.emtreeProtein expressionen_US
dc.subject.emtreeProtein functionen_US
dc.subject.emtreeProtein synthesisen_US
dc.subject.emtreeAnimalen_US
dc.subject.emtreeAnimal diseaseen_US
dc.subject.emtreeBlooden_US
dc.subject.emtreeDisease modelen_US
dc.subject.emtreeDog diseaseen_US
dc.subject.emtreeEscherichia colien_US
dc.subject.emtreeImmunologyen_US
dc.subject.emtreeMicrobiologyen_US
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