Please use this identifier to cite or link to this item: http://hdl.handle.net/11452/33709
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dc.contributor.authorŞeker, İsmail-
dc.date.accessioned2023-08-31T12:45:50Z-
dc.date.available2023-08-31T12:45:50Z-
dc.date.issued2017-
dc.identifier.citationGül, Ö. Ö. vd. (2017). ''Correlates of visceral and subcutaneous fat thickness in non-diabetic obese and morbidly obese patients''. Acta Medica Mediterranea, 33(6), 1017-1023.en_US
dc.identifier.issn0393-6384-
dc.identifier.issn2283-9720-
dc.identifier.urihttps://doi.org/10.19193/0393-6384_2017_6_161-
dc.identifier.urihttps://www.actamedicamediterranea.com/archive/2017/medica-6/correlates-of-visceral-and-subcutaneous-fat-thickness-in-non-diabetic-obese-and-morbidly-obese-patients-
dc.identifier.urihttp://hdl.handle.net/11452/33709-
dc.description.abstractBackground: To determine the correlates of visceral and subcutaneous fat thickness in non-diabetic obese and morbidly obese patients Methods: A total of 31 obese female outpatients composed of morbidly obese (n=16, BMI of >= 40kg/m(2)) and obese (n=15, BMI of 30-39.9kg/m(2)) patients were included in the present study. Data on age, anthropometrics, blood biochemistry, HOMA-IR, carotid intima-media thickness (CIMT) were recorded in each subject as were plasma resistin (mu g/L) and visfatin (mu g/ml) levels, epicardial, subcutaneous and abdominal fat thickness (mm). Correlates of visceral and subcutaneous fat thickness were determined via linear regression models with inclusion of severity of obesity, insulin resistance, plasma resistin and visfatin levels and CIMT as variables. Results: Epicardial fat thickness (mm) was 3.1(1.0-10.20) and 8.8(2.60-13.0), CIMT (mm) was 5.8(4.7-8.9) and 5.9(4-8.6), abdominal fat thickness (mm) was 10.8(7.8-16.1) and 13.2(8.7-16.5), subcutaneous fat thickness(mm) was 43.8(28.4-62.9) and 57.4(39.5-72.7), plasma resistin levels (mu g/L) were 8.5(4.7-38.1) and 10.8(0.7-26.4) and plasma visfatin levels (mu g/ml) were 55.5(5.1-209.5) and 78.2(4.7-228) in obese and morbidly obese patients, respectively. Linear regression analysis revealed that being morbidly obese was likely to increase epicardial fat thickness by 4.33mm(p=0.004) compared with obesity, while for each 1 unit increase in HOMA levels, subcutaneous fat thickness was likely to decrease by 1.16mm(p=0.009). Conclusion: In conclusion, our findings revealed that neither plasma levels for resistin and visfatin nor CIMT correlated with visceral or subcutaneous fat thickness in non-diabetic obese females, while increase in subcutaneous and epicardial fat thickness values were noted with decrease in HOMA-IR and the presence of morbid obesity, respectively.en_US
dc.language.isoenen_US
dc.publisherCarbone Editoreen_US
dc.rightsinfo:eu-repo/semantics/closedAccessen_US
dc.subjectGeneral & internal medicineen_US
dc.subjectObesityen_US
dc.subjectVisceral faten_US
dc.subjectSubcutaneous faten_US
dc.subjectVisfatinen_US
dc.subjectResistinen_US
dc.subjectInsulin resistanceen_US
dc.subjectEpicardial adipose-tissueen_US
dc.subjectCoronary-artery-diseaseen_US
dc.subjectIntima-media thicknessen_US
dc.subjectPlasma resistin levelsen_US
dc.subjectInsulin-resistanceen_US
dc.subjectInflammatory mediatorsen_US
dc.subjectVisfatin levelsen_US
dc.subjectHeart-diseaseen_US
dc.subjectAtherosclerosiseen_US
dc.subjectXpressionen_US
dc.titleCorrelates of visceral and subcutaneous fat thickness in non-diabetic obese and morbidly obese patientsen_US
dc.typeArticleen_US
dc.identifier.wos000411949500020tr_TR
dc.identifier.scopus2-s2.0-85029208615tr_TR
dc.relation.publicationcategoryMakale - Uluslararası Hakemli Dergitr_TR
dc.contributor.departmentUludağ Üniversitesi/Tıp Fakültesi/Endokrinoloji ve Metabolizma Anabilim Dalı.tr_TR
dc.contributor.departmentUludağ Üniversitesi/Tıp Fakültesi/Biyoistatistik Anabilim Dalı/İç Hastalıkları Anabilim Dalı.tr_TR
dc.contributor.departmentUludağ Üniversitesi/Fen-Edebiyat Fakültesi/Kardiyoloji Anabilim Dalı.tr_TR
dc.contributor.orcid0000-0002-2593-7196tr_TR
dc.contributor.orcid0000-0001-8404-8252tr_TR
dc.identifier.startpage1017tr_TR
dc.identifier.endpage1023tr_TR
dc.identifier.volume33tr_TR
dc.identifier.issue6tr_TR
dc.relation.journalActa Medica Mediterraneaen_US
dc.contributor.buuauthorGül, Özen Öz-
dc.contributor.buuauthorPekgöz, Murat-
dc.contributor.buuauthorGüllülü, Sümeyye-
dc.contributor.buuauthorCander, Soner-
dc.contributor.buuauthorTutuncu, Ahmet-
dc.contributor.buuauthorSağ, Saim-
dc.contributor.buuauthorAçıkgöz, Ebru-
dc.contributor.buuauthorSarandol, Emre-
dc.contributor.buuauthorErsoy, Canan-
dc.contributor.researcheridAAH-8861-2021tr_TR
dc.contributor.researcheridABE-1716-2020tr_TR
dc.contributor.researcheridAAW-9185-2020tr_TR
dc.contributor.researcheridAAI-1005-2021tr_TR
dc.relation.collaborationSanayitr_TR
dc.subject.wosMedicine, general & internalen_US
dc.indexed.wosSCIEen_US
dc.wos.quartileQ4en_US
dc.contributor.scopusid26040787100tr_TR
dc.contributor.scopusid36010142900tr_TR
dc.contributor.scopusid57204660708tr_TR
dc.contributor.scopusid25027068600tr_TR
dc.contributor.scopusid7004229025tr_TR
dc.contributor.scopusid12140008100tr_TR
dc.contributor.scopusid56328183700tr_TR
dc.contributor.scopusid55943324800tr_TR
dc.contributor.scopusid6701485882tr_TR
dc.subject.scopusFat Thickness; Adipose Tissues; Coronary Artery Diseaseen_US
dc.subject.emtreeNicotinamide phosphoribosyltransferaseen_US
dc.subject.emtreeResistinen_US
dc.subject.emtreeAbdominal faten_US
dc.subject.emtreeAdulten_US
dc.subject.emtreeAgeen_US
dc.subject.emtreeAnthropometryen_US
dc.subject.emtreeArterial wall thicknessen_US
dc.subject.emtreeArticleen_US
dc.subject.emtreeBody massen_US
dc.subject.emtreeClinical articleen_US
dc.subject.emtreeCorrelation analysisen_US
dc.subject.emtreeDisease severityen_US
dc.subject.emtreeEpicardial faten_US
dc.subject.emtreeFemaleen_US
dc.subject.emtreeHumanen_US
dc.subject.emtreeInsulin resistanceen_US
dc.subject.emtreeIntraperitoneal faten_US
dc.subject.emtreeMorbid obesityen_US
dc.subject.emtreeObesityen_US
dc.subject.emtreeProtein blood levelen_US
dc.subject.emtreeSubcutaneous faten_US
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